VNO is a
non functioning organ, therefore I believe that the hypothsis you mentioned above is incorrect
Au
Contraire! I'd like to see your evidence bronzi boy.. While the VNO is less of a driving force in higher
mammels (such as are we) it nontheless influences behavior - this is common knowledge.
There is also the fact
that physiological reaction (behavior) to a vomeropherin (such as in Androstadienone [or A1]) strongly correlates
its activity at the VNO. Same story with Estratetraenol.
AndrostAnone is known to evoke a
stronger response in the human female VNO then does androstenone (thus proving my original theory correct).
See
for yourself:
AndrostAnone is the first bar
shown (5α-androstan-3-one)
Androstenone (-none) is the
forth one listed (5α-androst-16-en-3-one)
Androstadienone is the fifth one listed
(androsta-4,16-en-3-one)
P.S. There is also
the fact that androstAnone is far more volatile than androstenone. AndrostAnone's MP is 103˚C,
whereas androstenone's MP is 145˚C (according to the Sigma-Aldrich catalog). Thus, the vapor
pressure is higher and hence it evaporates into the air (where pheromones are active) much moreso than
androstenone.
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